Лечение простатита и повышение потенции

ivanov824 писал(а):А может быть так что хламилии пцр спермы не определились,а пцр сока простаты определились.

Kepler писал(а):Сходи сдай анализы в другой лабе, мне кажеться что тебе доктор голову дурит. Нет у тебя ни хламидиоза, ни трихомониаза. Если у человека есть ИППП они должны определяться прямо из уретры, ПЦР настолько чувствительный анализ, что способен уловить даже одну молекулу любого вещества и для результата достаточно обычного мазка!

А если ты сдашь бак.посев и у тебя опять определят Enterococcus faecalis, то именно он и будет виновником твоего простатита.

Enterococcus faecalis являеться причиной порядка 73% воспалительных заболеваний мочеполовой системы.

Abstract
Trichomonas vaginalis is a common sexually transmitted protozoan parasite. Although often considered simply a nuisance infection, T. vaginalis has been implicated in premature rupture of placental membranes and increases in the risk of acquiring human immunodeficiency virus. Metronidazole, a 5-nitroimidazole, is currently the drug of choice to treat T. vaginalis infection. Because some patients have severe reactions to metronidazole and others are infected with metronidazole-resistant T. vaginalis, we were prompted to investigate alternative therapies. Tinidazole, another 5-nitroimidazole used in other countries to treat T. vaginalis infections, and furazolidone, a nitrofuran presently used to treat giardiasis and infections with some anaerobic enteric bacteria, were investigated for effectiveness against 9 metronidazole-susceptible and 12 metronidazole-resistant T. vaginalis patient isolates. The in vitro aerobic and anaerobic minimum lethal concentrations (MLC) and the time for drug efficacy were determined. Tinidazole killed the metronidazole-susceptible isolates at a low MLC but was effective against only 4 of the 12 metronidazole-resistant isolates. In contrast, furazolidone was effective at a low MLC for all isolates. When tinidazole was effective, it required > 6 h to kill trichomonads. However, furazolidone killed both metronidazole-susceptible and resistant trichomonads within 2 to 3 h of exposure. These data suggest that furazolidone may be a good candidate for treating metronidazole-resistant trichomoniasis and that further investigation of this drug is warranted.

Abstract
An in vitro study was carried out on ten first clinical isolates of Trichomonas vaginalis. Increasing levels of butaconazole were added to 150 microliters of a dilution in Roiron medium adjusted to 50,000 Trichomonas/ml. Results were read after 1, 2, 6 and 24 hours aerobic incubation at 37 degrees C. Minimum lethal concentration was 93 micrograms/microliters after one hour, 23 micrograms/microliter after six hours and 12 micrograms/microliters after 24 hours. These findings warrant in vivo studies of butoconazole against Trichomonas. This drug, which has already been proved effective in fungal infections, may prove very useful since concomitant infection of the genital tract by Trichomonas and Candida is common.

Abstract
Trichomonas vaginalis (T. vaginalis) is a flagellated protozoan commonly causing sexually transmitted disease. T. vaginalis infections are treated with a 5-nitroimidazole derivate. However, drug resistance has been known to occur for a long time and new alternatives are under investigation. Arbutus unedo is a wild plant mainly growing in maquis and rocky places of the seaboard in Southern Europe. In our study, ethanolic, water, hexane and ethyl acetate extracts of Arbutus unedo leaves were tested in vitro against T. vaginalis trophozoites and the ethyl acetate extract of Arbutus unedo leaves was found to be effective (Growth inhibition rate (GI): 100%, at the concentration of 500 microg/ml). It may be a promising anti-trichomonacidal agent in the future and further experiments are needed.

Abstract
Cultured T. vaginalis was used for the anti-trichomonas test at different times, concentrations of propolis and densities of the parasites. After being cultured for 0, 6, 12, 24 hrs, the survival rate of the parasites was (91.50+/-3.11)%, (43.00+/-6.83)%, (22.25+/-5.32)% and (11.50+/-5.74)% respectively with a significant difference between propolis group and the control. Under the concentrations of 0.24, 0.48, 0.96, 1.92, 3.84 and 7.68 (mg/ml) with 24 hrs culture, the survival rate was (88.00+/-5.29)%, (92.67+/-4.16)%, (90.0+/-6.00)%, (84.00+/-4.00)%, (2.67+/-1.15)%, and 0 respectively. The results showed that propolis possesses clear in vitro anti-trichomonas activity which is relevant to the duration of culture and the concentration of the agent.

Родион писал(а):Тоже думал что хронический простатит у меня. Года 2 его лечил, кучу анализов сдавал, иппп нету не одной, а простатит есть. До этого лечил трихи и уреаплазму, думал вылечил. Нашли зимой в сперме триху. ПЦР ее вообще не определяет, только микроскопическим исследование лаборант находит. Можно конечна подумать, что лаборант принимает за триху что-то другое, но лечение то трихи помогает. Не какого простатита нету пока лечишь, только триха эта не дохнет и через недельку снова все появляется. Парни, это я к тому что если кто болел трихой, а птом у него простатит остался и лечение не помогает, сдайте на микроскопическое исследование сперму, может у вас та-же ситуация что у меня.